Cytomegalovirus (CMV) infection induces an angiogenic response through hepatic stellate cells (HSCs) and leads to early post-transplant liver fibrosis (LF) and poor graft survival

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dc.contributor.authorOzdemir, B. H.
dc.contributor.authorOzgun, G.
dc.contributor.authorSoy, Ayvazoglu E. H.
dc.contributor.authorHaberal, N.
dc.contributor.authorMoray, G.
dc.contributor.authorHaberal, M.
dc.date.accessioned2020-10-15T07:16:17Z
dc.date.available2020-10-15T07:16:17Z
dc.date.issued2019
dc.description.sponsorshipInt Liver Transplantat Socen_US
dc.identifier.endpage61en_US
dc.identifier.issn0041-1337en_US
dc.identifier.issue8en_US
dc.identifier.startpage60en_US
dc.identifier.urihttp://hdl.handle.net/11727/4904
dc.identifier.volume103en_US
dc.identifier.wos000494805000099en_US
dc.language.isoengen_US
dc.relation.journalTRANSPLANTATIONen_US
dc.rightsinfo:eu-repo/semantics/closedAccessen_US
dc.titleCytomegalovirus (CMV) infection induces an angiogenic response through hepatic stellate cells (HSCs) and leads to early post-transplant liver fibrosis (LF) and poor graft survivalen_US
dc.typeconferenceObjecten_US

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