TR-Dizin İndeksli Açık & Kapalı Erişimli Yayınlar
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Item 2020 ESC Core Curriculum for the Cardiologist: What has changed? Why? Should we also change our curriculum?(2020) Yildirir, Aylin; 0000-0001-8750-5287; 33257609; 33257609; A-4947-2018Item Abdominopelvic Tumor Implants Detected with a Bone Scan in a Case of Borderline Ovarian Tumor(2015) Gencoglu, Esra Arzu; Cinar, Alev; Aktas, Aysel; 0000-0002-3426-2987; 0000-0003-4631-1683; ABA-7670-2021; ABG-1864-2020Item Acquired pseudoaneurysm of the sinus of Valsalva(2017) Coner, Ali; Akinci, Sinan; Cicek, Davran; Saba, Tonguc; Muderrisoglu, Haldun; 0000-0001-5250-5404; 0000-0002-9635-6313; 0000-0002-5711-8873; 28902654; AAD-5564-2021; AAG-8233-2020; ABD-7321-2021; AAC-8036-2020Item Amyloid Goiter Due to Familial Mediterranean Fever in a Patient with Byler Syndrome: A Case Report(2014) Cigerli, Ozlem; Unal, Asli Dogruk; Parildar, Hulya; Demiralay, Ebru; Tarcin, Ozlem; 25337425Background: Familial Mediterranean Fever (FMF), also inherited with autosomal recessive trait, is characterized by recurrent episodes of fever, arthritis, and serositis. Congenital Byler Syndrome (Progressive Familial Intrahepatic Cholestasis) inherited with autosomal recessive trait and characterized by defective secretion of bile acids. FMF associated Amyloid A deposition occurs in many tissues and organs, but amyloid goiter is a rare entity that leads to enlargement and dysfunction of the thyroid. Case Report: We present a rare case of 24 year old male patient who had liver and kidney transplantation due to Byler Syndrome and secondary amyloidosis related to FMF, diagnosed as rapidly growing large amyloid goiter. Deposits of extracellular amyloid and dense adipose metaplasia diagnostic for amyloid goiter are determined upon histopathological examination of thyroidectomy material. Conclusion: When goiter was detected in cases with history of systemic amyloidosis and rapidly growing goitre, amyloid goiter should be remembered at first. This case is unique since two autosomal genetic disorders are together in the same patient and important as it emphasizes the consequences of consanguineous marriage, early diagnosis and treatment compliance of FMF and the awareness of amyloid goiter in patients followed by primary care physicians and healthcare professionals.organizational unit.listelement.badge Cardiac biomarkers in coronary slow flow: Endocan and omentin-1(2019) Okyay, Kaan; 31219435; AAK-7355-2020Item Combined catheter thrombus fragmentation and percutaneous thrombectomy in a patient with massive pulmonary emboli and acute cerebral infarct(2015) Ugurlu, Aylin Ozsancak; Cinar, Ozlem; Caymaz, Ismail; Cevik, Halime; Gumus, Burcak; 25550253Item Endovascular Diagnosis and Successful Treatment of Massive Gastrointestinal Hemorrhage in Children(2018) Temiz, Abdulkerim; Gedikoglu, Murat; Ezer, Semire Serin; Oguzkurt, Pelin; Hicsonmez, Akgun; 29666037Item Extraordinary biliary variant(2017) Suna, Nuretdin; Etik, Digdem Ozer; Ocal, Serkan; Selcuk, Haldun; Hilmioglu, Fatih; Boyacioglu, Sedat; 0000-0003-3719-9482; 0000-0002-9370-1126; 0000-0002-6440-5686; 0000-0002-4724-0728; 0000-0002-8445-6413; 0000-0001-6234-7788; 28336501; ABH-4817-2020; AAE-7637-2021; AAJ-4437-2021; AAJ-4707-2021; AAJ-6976-2021; AAI-8822-2021Item Galectin-3 for risk stratification in acute coronary syndrome(2020) Okyay, Kaan; 0000-0001-6134-8826; 33257618; AAK-7355-2020Item Hiccups in an Adult Case with Schizophrenia due to Aripiprazole: A Case Report(2021) Kutuk, Meryem Ozlem; Berdzenishvili, Ekaterina; Aksu, Gulen Guler; 33795958Neurotransmitters and neuroreceptors involved in the pathophysiology of hiccups are not well defined. However, dopamine and serotonin are reported to have roles in activating hiccups, and recent case reports suggest that some psychopharmacologic medications -such as antipsychotics- may trigger hiccups in many cases. Our case describes the activation of hiccups in a young male with schizophrenia while being treated with aripiprazole. The patient was switched from risperidone to aripiprazole due to excessive sedation, hiccups started within 48 hours of initiation of treatment with aripiprazole at a dosage of 15 mg/day, and no change in the hiccups was observed despite a dose reduction. Discontinuation of aripiprazole treatment resulted in complete relief from hiccups. This case report shows that antipsychotics may trigger hiccups.Item Perioperative planning in the COVID-19 pandemic: Vascular issues(2020) Akay, Tankut; 0000-0003-1581-5352; 32551152Item Pleiotropic effects of statins: New evidences(2021) Okyay, Kaan; 34623295Item Predictors of Pain During Cystoscopy(2015) Ipekci, Tumay; Akin, Yiğit; Hoscan, Mustafa BurakItem Pseudotumoral Cystitis Cystica of the Bladder(2015) Gul, Umit; Cystica, CystitisItem A rare cause of gastric outlet obstruction(2017) Suna, Nurettin; Ocal, Serkan; Etik, Digdem Ozer; Selcuk, Haldun; Hilmioglu, Fatih; Boyacioglu, Sedat; 0000-0001-6234-7788; 0000-0003-3719-9482; 0000-0002-8445-6413; 0000-0002-4724-0728; 0000-0002-6440-5686; 0000-0002-9370-1126; 28705787; AAI-8822-2021; ABH-4817-2020; AAJ-6976-2021; AAJ-4707-2021; AAJ-4437-2021; AAE-7637-2021Item A Rare Cause of Hemifacial Spasm: Papillary Oncocytic Cystadenoma(2016) Erol, Ozan; Aydin, Erdinc; 0000-0001-6864-7378; 27761290; AAJ-2379-2021Background: Hemifacial spasm is a sudden, involuntary and synchronous spasm of the facial muscles. The most frequent cause of this condition is compression of the facial nerves due to vascular pathologies. The most commonly used method of treatment is Botulinum toxin injection. However, the gold standard treatment is surgical treatment. Case Report: A 64-year-old male patient with hemifacial spasms, which had occurred due to a rare parotid mass that had been surgically treated, is presented in this case. Conclusion: This case report demonstrates that long-standing parotid gland masses may compress the facial nerves and cause demyelination in the nerve and thus may cause spasms in the facial muscles.Item A Rare Cause of Left Ventricular Mass: Cardiac Hemangioma(2018) Altin, Cihan; Gullu, Hakan; Bilgel, Ziya Gokalp; Yilmaz, Mustafa; Canpolat, Tuba; Gulcan, Omer; 29588260Item A Rare Complication of Cholecystitis Diagnosed by Ultrasonography: Gallbladder Perforation(2015) Ergun, Tarkan; Lakadamyali, HaticeItem A Report of Brugada Syndrome Presenting with Cardiac Arrest Triggered by Verapamil Intoxication(2017) Erdogan, Ilkay; Yakut, Kahraman; Varan, Birgul; Atar, Ilyas; 0000-0001-6887-3033; 0000-0002-6719-8563; 29215340; ABB-2220-2021; AAJ-2305-2021; ABB-1767-2021Background: Brugada syndrome is a disease characterized by a specific electrocardiographic pattern and an increased risk of sudden cardiac death. We present this case with the updated literature to emphasise the need to consider the diagnosis of Brugada syndrome in patients admitted to the emergency ward with sudden cardiac arrest. Case Report: A 16-year-old female patient was admitted to the emergency ward with complaints of weakness and abdominal pain, and she had four cardiac arrests during her evaluation period. She was referred to our clinic for permanent pacemaker implantation. She was on a temporary pace maker after having had C-reactive protein. Her physical exam was normal except for bilaterally decreased lung sounds. Lung x-ray and computed tomography, which were performed by another institution, revealed minimal pleural effusion and nothing else of significance. Blood and peritoneal fluid samples were sterile. Echocardiographic exam and cardiac enzymes were also in the normal ranges. Electrocardiographic showed incomplete right branch block in leads V1 and V2. An ajmaline test revealed specific electrocardiographic findings of the type I Brugada pattern. We proposed implanting an implantable cardioverter defibrillator to the patient as there were positive findings on the ajmaline test as well as a history of sudden cardiac arrest. After this treatment proposal, the patient's family admitted that she had taken a high dose of verapamil and thus, the encountered bradycardia was associated with verapamil overuse. The ajmaline test was repeated as it was contemplated that the previous positive ajmaline test had been associated with verapamil overuse. Implantable cardioverter defibrillator implantation was proposed again as there was a history of sudden cardiac arrest; however, the family did not consent to implantable cardioverter defibrillator, and the patient was discharged and followed up. Conclusion: Brugada syndrome should be considered for patients who are admitted to the emergency ward with sudden cardiac arrest though surface electrocardiographic is normal. If there is a suspicion of Brugada syndrome, repeated electrocardiographic should be performed on different occasions. Diagnosis can be clarified by upper costal electrocardiographic or by administering Na channel blockers during electrocardiographic performance.