Wos Açık Erişimli Yayınlar

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Author: search.filters.author.Ozkan, Muratsearch.filters.applied.f.publicationcategory: search.filters.publicationcategory.Makale - Ulusal Hakemli Dergi

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    Infective Endocarditis Caused by Abiotrophia defectiva in a Pediatric Patient
    (2021) Ozkan, Murat; 0000-0003-3991-8479
    Abiotrophia defectiva is a nutritional defective streptococcus that is difficult to grow in routine culture media, is found in the oral mucosa, gastrointestinal and urogenital system flora and is a rare cause of infective endocarditis. An 11-year-old boy with underlying operated congenital heart defect presented with prolonged fever. Abiotrophia defectiva is yielded in the blood cultures taken properly with a pre-diagnosis of infective endocarditis. Although there was no vegetation or other finding of endocarditis on echocardiography, surgery decision was made because bacteria continued to grow in the blood culture under appropriate antibiotic treatment. During surgery, it was observed that the contegrapulmonic valved conduit was severely degenerated and calcified. A new contegrapulmonic valve conduit replacement was performed. Serious complications occur more frequently in infective endocarditis caused by Abiotrophia defectiva compared to other etiogic agents of infective endocarditis. With this case, we wanted to emphasize that in Abiotrophia defectiva infective endocarditis, the possibility of antibiotic treatment failure and surgical procedure requirement is quite high.
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    A case of neonatal arterial thrombosis mimicking interrupted aortic arch
    (2015) Gursu, Hazim Alper; Varan, Birgul; Oktay, Ayla; Ozkan, Murat; 26265897
    Neonatal arterial thrombosis is a very rare entity with clinical findings resembling coarctation of aorta or interrupted aortic arch. A two day-old male newborn was admitted to another hospital with difficulty in sucking and sleepiness. On echocardiographic examination, a diagnosis of interrupted aortic arch was made and he was treated with prostoglandin E2. On physical examination, on admission to our center, the feet were bilaterally cold. The pulses were not palpable and there were ecchymotic regions in the lower extremities. Echocardiography ruled out interrupted aortic arch. Computerized tomographic angiography revealed a large thrombosis and total occlusion of the abdominal aorta. Since there was no response to treatment with tissue plasminogen activator, we performed thrombectomy. Homozygous Factor V Leiden and Methylenetetrahydrofolate reductase mutations were found in this patient. Neonatal aortic thrombosis which is observed very rarely but which is fatal should be considered in the differential diagnosis of coarctation of aorta and interrupted aortic arch.
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    Case report and the surgical treatment of two cases with pulmonary atresia in which pulmonary arteries is circulated by coronary arteries
    (2018) Tokel, Kursad; Yakut, Kahraman; Varan, Birgul; Erdogan, Ilkay; Ozkan, Murat; 0000-0002-6719-8563; 0000-0001-6887-3033; 0000-0002-6759-1795; 29664428; ABB-1767-2021; AAJ-2305-2021; ABB-2220-2021; AAF-3253-2021
    Pulmonary atresia (PA) and ventricular septal defect (VSD) can occur in a variety of ways, from simple valve atresia to a condition in which circulation to the pulmonary bed occurs through collateral arteries separated from the aorta and there are no real pulmonary arteries, or they are present but hypoplastic. The size of the pulmonary arteries and concomitant complex cardiac lesions are important in making decisions about treatment and correctional alternatives. While complete correction surgeries in the style of a correction of tetralogy of Fallot are performed in simpler cases, many very invasive procedures are also performed and the resulting quality of life is very variable. The size of the pulmonary artery and its branches and the presence of accompanying collateral vessels are determining factors in the management of the disease. In this report, 2 cases of VSD and PA, in which the circulation of the pulmonary arteries was through the coronary arteries, diagnosed as a result of echocardiography performed following murmurs heard during examination, are described. The first patient was 40 days old and the second was 2 days old. In the second case, diagnosis was confirmed by catheter angiography, and in the first case, the final diagnosis was made during surgery. The aim of this report is to emphasize the importance of pulmonary artery size, the presence of pulmonary confluence, the origin of circulation, and concomitant collateral arteries when considering treatment methods.