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dc.contributor.authorYilmaz, Hatice Eylul Bozkurt
dc.contributor.authorYilmaz, Mustafa
dc.contributor.authorSen, Nazan
dc.contributor.authorUnsal, Zuhal Ekici
dc.contributor.authorEyuboglu, Fusun Oner
dc.contributor.authorAkcay, Sule
dc.date.accessioned2021-02-26T08:49:53Z
dc.date.available2021-02-26T08:49:53Z
dc.date.issued2019
dc.identifier.issn1302-7808en_US
dc.identifier.urihttps://turkthoracj.org/en/investigation-of-the-relationship-between-asthma-and-visceral-obesity-by-epicardial-fat-thickness-measurement-131049
dc.identifier.urihttp://hdl.handle.net/11727/5425
dc.description.abstractOBJECTIVES: Obesity is a risk factor defined in recent years for asthma. It is associated not only with asthma but also with many cardiovascular diseases. Visceral obesity is a more significant risk factor than general obesity in cardiovascular diseases. Although the association of visceral obesity with cardiovascular diseases is well known, the relationship in patients with asthma is not fully understood. The aim of the present study was to investigate whether there is a relationship between asthma and visceral fat by using epicardial fat thickness (EFT) measurement. MATERIALS AND METHODS: A total of 401 subjects (229 patients with persistent asthma and 172 controls) were enrolled in the study. In our study, EFT was measured, recorded by echocardiography, and was evaluated whether there was a statistical significant difference between the two groups. RESULTS: The mean EFT was 5.84 +/- 0.79 mm in the patient group and 5.71 +/- 0.93 mm in the control group. There was no statistically significant difference between the groups (p=0.145). Similarly, when we compared control and asthma severity subgroups, we did not find statistically significant differences (control group mean 5.71 +/- 0.93 mm, mild group mean 5.86 +/- 0.81 mm, moderate group mean 5.8 +/- 0.84 mm, and severe group mean 5.83 +/- 0.67 mm, p=0.505). CONCLUSION: In the present study, we observed that the EFT did not increase in patients with asthma compared with the normal population. Based on our results, we suggest that visceral obesity may not be a significant risk factor for asthma.en_US
dc.language.isoengen_US
dc.relation.isversionof10.5152/TurkThoracJ.2018.18028en_US
dc.rightsinfo:eu-repo/semantics/openAccessen_US
dc.subjectAsthmaen_US
dc.subjectallergyen_US
dc.subjectclinical problemsen_US
dc.subjectdiagnostic methodsen_US
dc.titleInvestigation of the Relationship between Asthma and Visceral Obesity by Epicardial Fat Thickness Measurementen_US
dc.typearticleen_US
dc.relation.journalTURKISH THORACIC JOURNALen_US
dc.identifier.volume20en_US
dc.identifier.issue1en_US
dc.identifier.startpage1en_US
dc.identifier.endpage5en_US
dc.identifier.wos000459396200001en_US
dc.identifier.scopus2-s2.0-85059589107en_US
dc.contributor.pubmedID30664419en_US
dc.contributor.orcID0000-0003-3225-2686en_US
dc.contributor.orcID0000-0002-8360-6459en_US
dc.contributor.orcIDAAB-5175-2021en_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergien_US
dc.contributor.researcherIDAAR-4338-2020en_US
dc.contributor.researcherIDAAD-5602-2021en_US


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